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For example, oxidative stress-induced microRNA-9/9* has been implicated in the MELAS phenotype, while downregulation of microRNA-181a/b, which promotes MQC through the activation of mitochondrial biogenesis and mitophagy, has shown protective effects on RGCs in an LHON mouse model

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But the peptide has been shown to have therapeutic healing effects in both bone and tendon injury models (28, 29, 55)